Molecule

Eosinophils

Effector granulocytes

Expression change
Massively infiltrated in tissue
Evidence level
established
Targeted by
Mepolizumab, Benralizumab

Role in pathogenesis

Tissue eosinophilia is the hallmark of CRSwNP. Eosinophils release MBP, ECP, EDN, and EPO — cytotoxic proteins that damage epithelium and perpetuate inflammation. Eosinophil counts in polyps correlate with recurrence.

Targeting drugs (2)

DrugMechanismResponse rateLine
MepolizumabAnti–IL-5 monoclonal antibodySignificant NPS reduction1st-line biologic
BenralizumabAnti–IL-5Rα monoclonal antibody (ADCC-mediated eosinophil depletion)Near-complete eosinophil depletion1st-line biologic

Sources (2)

P2Bachert C et al. (2015) Endotype-driven care pathways in patients with chronic rhinosinusitisDOI
P5Takeda K et al. (2024) Eosinophil contribution to tissue remodelling in CRSwNPDOI