IL-1β

Central pro-inflammatory cytokine driving systemic inflammation

Expression
Elevated
Evidence level
established
Targeted by
Anakinra, Canakinumab

Role in pathogenesis

IL-1β is a key effector cytokine in AOSD, produced by NLRP3 inflammasome-mediated caspase-1 cleavage of pro-IL-1β. Drives fever, arthritis, rash, and acute-phase response. The dramatic efficacy of IL-1 blockade (anakinra, canakinumab) confirms its central pathogenic role. Both anakinra and canakinumab have received EMA approval for AOSD based on IL-1β's established role.

Targeting drugs (2)

DrugMechanismResponseLine
AnakinraIL-1 receptor antagonist~80-90%2nd
CanakinumabAnti-IL-1beta monoclonal antibody~67%2nd

Sources (3)

DetailsFeist E et al. (2018) Mechanisms, biomarkers and targets for adult-onset Still's disease · Nat Rev RheumatolPubMed
DetailsChen DY et al. (2017) Elevated expression of the NLRP3 inflammasome and its correlation with disease activity in adult-onset Still disease · J RheumatolPubMed
DetailsNordstrom D et al. (2012) Beneficial effect of interleukin 1 inhibition with anakinra in adult-onset Still's disease: an open, randomized, multicenter study · J RheumatolPubMed